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CHPC - Research Computing and Data Support for the University

In addition to deploying and operating high performance computational resources and providing advanced user support and training, CHPC serves as an expert team to broadly support the increasingly diverse research computing and data needs on campus. These needs include support for big data, big data movement, data analytics, security, virtual machines, Windows science application servers, protected environments for data mining and analysis of protected health information, and advanced networking.

If you are new to CHPC, the best place to start to get more information on CHPC resources and policies is our Getting Started page.

Upcoming Presentations:


Posted September 8th, 2023

CHPC is reaching out to remind our users of their responsibility to understand what the software being used is doing, especially software that you download, install, or compile yourself. Read More...


Posted August 23, 2023

General Environment FastX Outage Monday August 21, 2023 starting at 9am

Posted August 11, 2023

Allocation Requests for Fall 2023 are Due September 1st, 2023

Posted August 1, 2023

CHPC ANNOUNCEMENT:  Establishing quotas on /scratch/general/vast effective August 1, 2023

Posted July 5, 2023

FastX3 Outage - Saturday, July 1st, license issue resolved by 7:45pm

Posted July 3, 2023

CHPC Downtime: Tuesday July 11, 2023 starting at 7:30 am

Posted June 26, 2023

Spring 2023 CHPC Newsletter

Posted April 19th, 2023

CHPC ANNOUNCEMENT: CHPC staff working both remotely and hybrid schedules. 

News History...


Nanopore sequencing reveals rapid evolution of poxvirus genome structure driven by host-pathogen conflict

By Tom Sasani, Kelsey Rogers-Cone, Ryan Layer, Nels Elde & Aaron R. Quinlan

 Quinlan Lab, Department of Human Genomics and USTAR Center for Genetic Discovery, University of Utah

Vaccinia virus (VACV) encodes two host-range factors, E3L, and K3L, that each disrupt key antiviral host defenses. In the absence of E3L, VACV has been shown to rapidly adapt by duplicating K3L in tandem arrays, which confers a significant increase in fitness. Additionally, viruses accumulate H47R mutations within K3L, which provides an added fitness benefit. In order to investigate the relationship between K3L copy number amplification and H47R allele fixation, we sequenced VACV populations with the Oxford Nanopore (ONT) single-molecule platform. We discovered that H47R progressively accumulates within arrays of the duplicated K3L gene, and uncovered some of the interplay between allelic diversity and structural variation during viral evolution.

System Status

General Environment

last update: 2023-09-26 02:11:05
General Nodes
system cores % util.
kingspeak 957/988 96.86%
notchpeak 2829/3212 88.08%
lonepeak 2892/3236 89.37%
Owner/Restricted Nodes
system cores % util.
ash 3612/3812 94.75%
notchpeak 16033/16908 94.82%
kingspeak 5400/5484 98.47%
lonepeak 416/416 100%

Protected Environment

last update: 2023-09-26 02:10:04
General Nodes
system cores % util.
redwood 40/552 7.25%
Owner/Restricted Nodes
system cores % util.
redwood 873/5980 14.6%

Cluster Utilization

Last Updated: 9/22/23